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Causes, treatment and prevention of enlarged pharyngeal follicles. Lymphoid follicles in the stomach what is it So that the follicles are reduced, and the inflammatory process in the throat passes


lymphoid tissues are viable lymphocytes that are conserved around the periphery of the explant. In the groups of these elements, mitoses are found on the 4-5th day of cultivation, and subsequently secondary lymphoid follicles are formed in their place.


In the formation of secondary lymphoid follicles in organ cultures, one can see a special type of connection between reticular cells and lymphocytes. In many cases, the lymphoid follicle forms around the characteristic spherical structures formed by the reticular cells.

In birds, B-cell differentiation occurs in the bursa, the folds of which contain lymphoid follicles with cortical and cerebral zones.

Reduction of lymphoid follicles and marginal zones in the spleen.

The spleen capsule is somewhat wavy, the trabeculae are thickened, hyalinized. The lumens of the central arteries are narrowed, their wall is homogeneous, hyalinized. In some cases, lymphoid follicles are reduced in number and volume, lymphocytes in them are preserved only in the form of a narrow belt around the central arteries. Pycnoform lymphocytes are visible in areas of the preserved lymphoid tissue.

The submucosa of the small and large intestine is sharply edematous, loosened, in most cases infiltrated by cellular elements with a significant number of plasma cells. The same edema is even more pronounced from the side of the stroma of the villi of the small intestine. Among the edematous tissue of the submucosa of the large intestine, there are perivascular hemorrhages (Fig. 15). The lymphoid follicles of the large intestine were not expressed. The epithelial cover in the superficial areas of individual villi and folds is necrotic, impregnated with fibrin, the cells are desquamated (Fig. 16). In the deep layers of the side

Subacute poisoning. Introduction / 5 from LD50 for 1 month causes a lag in body weight gain, depression of the central nervous system, anemia and an increase in the content of methemoglobin in the blood. Histologically, there is parenchymal dystrophy in the liver, and hyperplasia of lymphoid follicles in the spleen.


In some rats killed at various times after the onset of dusting, few loose or more compact nodular accumulations of macrophages were found in the lungs, located in the lumens of the alveoli, in the interalveolar septa and in the perivascular and peribronchial lymphoid follicles. The protoplasm of macrophages sometimes looked cellular, was palely stained with eosin, and had a grayish tint. The nuclei in these cells were often absent. Sometimes, small gray dust particles could be seen in the protoplasm of lscrophages. In animals that are dusty for

Most experimental rats killed after 2 and 5 months. after the introduction of polyvinyl butyral, individual macrophages were visible in the lungs against the background of emphysema and plethora, scattered in the lumens of the alveoli between the cells of the peribronchial and perivascular lymphoid follicles. In the protoplasm of some macrophages, phagocytosed small black dust particles were found.

The described compact accumulations of macrophages in the lung tissue and in the peribronchial and perivascular lymphoid follicles were mainly found in experimental rats.

In surviving rats killed after 1–3 b and 9 months. after the introduction of substances, in the lungs under the pleura and on the incision there were point or larger, up to 2-3 mm in diameter, blue spots. Bifurcation lymph nodes were not sharply enlarged and stained blue. Under the pleura, in the lumens of the alveoli, in the interalveolar septa, and in the lymphoid follicles, a blue substance was found that was in the protoplasm of macrophages or was free (Fig. 5). In addition, in rats killed after 6 and 9 months. after intratracheal administration of fat-soluble pure blue anthraquinone b / m, individual accumulations of the substance located in the interalveolar septa were surrounded by a small number of elongated connective tissue cells. No changes were found in the rest of the internal organs during the administration of the substances.

The peribronchial lymphoid follicles are markedly enlarged; in their peripheral parts, reproduction of reticular cells is observed. Expanded lymphatic vessels with a picture of lymphostasis are also visible here. From the side of the bronchial epithelium, there are phenomena of a proliferative-destructive nature.

Starting from the 5-6th day of cultivation, the lymphoid tissue regenerates in the cortical region of the explants. As with lymph node explantation, Tai and thymic cultivation, regeneration occurs in the form of lymphoid follicle formation, often with characteristic enlightened centers. Such structures are characteristic of lymph nodes, but are not found in the intact thymus in vivo, which reflects the different immunological role of these organs. It is known that antigens do not penetrate into the thymus and no differentiation of antibody-forming cells occurs. At the same time, when the antigen is directly injected into the thymus, secondary follicles are formed in it and plasma cells appear.

Morphologically immunized cultures did not differ from non-immunized ones. As usual, in the first 4 days most of the lymphoid tissue was destroyed and the stroma was preserved. This was followed by regeneration with the formation of lymphoid follicles in the cortex. The medulla was poorly restored, and plasma cells were observed in immunized cultures as rarely as in non-immunized ones.

The huge population of the body's lymphocytes can be conditionally divided into sedentary and wandering lymphoid cells. Most of the lymphocytes circulate in the body with the flow of blood and lymph. At the same time, a significant number of lymphoid cells are localized in organs, being a component of lymph nodes, spleen, Peyer's patches, non-encapsulated lymphoid follicles (in the loose connective tissue of the mucous membranes and skin). The division of many lymphocytes into sedentary and wandering is not absolute. There is a constant redistribution between these two populations.

The white pulp consists of periarteriolar lymphoid clutches (PALMs), many of which contain lymphoid follicles. It is surrounded by a marginal zone filled with numerous macrophages, antigen-presenting cells, slowly recirculating B-lymphocytes and normal killer cells. The red pulp contains venous canals (sinusoids), separated by splenic cords. Blood enters the tissue of the spleen through the trabecular arteries, giving rise to branched central arterioles. Some of these arterioles end in the white pulp and feed the breeding centers and the marginal zone of the follicle, but most reach the marginal zone or areas adjacent to it. Some branches of arterioles go directly into the red pulp, ending in the splenic cords. From the venous sinusoids, blood is collected into the pulp veins, then into the trabecular veins and from them into the splenic vein.

Histological structure of the lymph node. The cortical (C), paracortical (P) and cerebral (M) regions are visible. The section is stained to reveal the localization of T cells. Most of them are in the paracortical region, and a certain amount is present in the multiplication center (CR) of the secondary lymphoid follicle, in the cortical region and brain cords (MT). (Photo courtesy of Dr. A. Stevens and Prof. J. Lowe.)

A single lymphoid follicle in the colon. This nodule of lymphoid tissue is located in the mucous membrane and in the submucosa of the intestinal wall arrow). (Photo courtesy of Dr. A. Stevens and Prof. J. Lowe.)

A dome-shaped projection formed by the intestinal mucosa in an area devoid of villi. The superficial epithelium in this region, called follicle-associated epithelium (FEA), contains M cells. In depth

Gastritis is one of the most common diseases. This disorder of the digestive tract has many varieties. Lymphoid gastritis is a rare type that affects only one percent of patients. The development of this disease has its own characteristics.

Description of the disease

Lymphoid, or follicular gastritis is a special type of pathology that affects the stomach. This form of the disease is characterized by the appearance of follicles - formations on the gastric mucosa as a result of the accumulation of lymphocytes.

It is generally accepted that this type of disease occurs against the background of chronic gastritis associated with the bacterium Helicobacter pylori. In this case, bacteria penetrate into the epithelial cells of the gastric mucosa, causing their damage.

Damaged cells cannot fully perform their functions. As a result, the normal activity of the digestive organ is disrupted, motor skills are reduced, and the secretory gland malfunctions.

The body's immune system, which is responsible for its defense, begins to send lymphocytic cells to the focus of infection. Lymphocytes are cells that produce antibodies to protect the body from pathogenic microbes.

As inflammation develops, lymphocytes accumulate in the damaged area and cause follicular formation. Follicles (rounded formations in the form of bubbles) must neutralize the harmful effects of microorganisms on the mucous layer. At the same time, they prevent the cells from secreting gastric juice, for normal digestion.

Follicles can grow to a considerable size and form a dense layer, interfering with the activity of the epithelial glands. As a result, the production of hydrochloric acid, which is necessary for food processing, is reduced.

Causes and symptoms

Lymphoid gastritis is not an independent disease. It occurs against the background of already existing pathologies in the epithelium of the mucous layer. In the overwhelming majority of cases, the inflammatory process in the stomach begins due to infection of the body with the bacterium Helicobacter pylori.

For the development of inflammation and the growth of microorganisms, appropriate conditions are necessary. The following factors can provoke the activation of bacteria in the body:

  • unhealthy diet, prolonged fasting, abuse of junk and fatty foods;
  • nervous experiences, strong psycho-emotional stress;
  • long-term premiere of alcoholic beverages, frequent smoking;
  • disorders of the autonomic system of the body.

The symptoms of follicular gastritis are similar to those of other forms of gastritis initiated by the Helicobacter bacterium. The main signs can be considered:

  • pain syndrome that manifests itself in the morning hours, as well as some time after eating;
  • sour belching, heartburn due to excess hydrochloric acid in the stomach;
  • decreased appetite;
  • dysfunction of the intestines, which is manifested by diarrhea and constipation.

With the development of an inflammatory process in the stomach, sensations of heaviness, distention and bloating appear. Nausea also appears, which is often accompanied by vomiting. Diarrhea can be followed by constipation, which happens quite often.

With an advanced form of the disease, the patient's condition worsens. Weakness, general body fatigue appears. Characterized by pallor and dryness of the skin, a white coating appears on the tongue. A decrease in appetite leads to a decrease in the patient's weight.

Diagnostics and treatment

Diagnosis of lymphofollicular gastritis is often difficult due to the specificity of the disease. This is due to the dim severity of the signs of this pathology. Visually, changes in the gastric mucosa may resemble atrophic or hyperplastic gastritis.

Rounded formations appear on the surface of the epithelium, which can take on different sizes. There is also an overgrowth of the mucous layer, similar to what happens with hyperplastic gastritis. Folds with erosive formations may appear on the inner surface of the stomach.

To make an accurate diagnosis, an endoscopy of the stomach is required, which consists in introducing a special probe with a video camera into the stomach cavity. This enables the doctor to see the development of pathology in the digestive organ and changes in the mucous layer.

In addition to endoscopy, a biopsy examination is performed. During a biopsy, tissues are taken from the patient's stomach and further analyzed in order to identify the cause of inflammation and determine the form of gastritis.

Also, the patient donates blood, urine and feces for analysis, which determine the deviation from the norm of various indicators and the presence of foreign impurities in the discharge.

In the event that lymphoid gastritis is diagnosed, the treatment is the same as for other types of gastritis. The treatment process consists of a set of measures. He suggests:

  • medicinal effect;
  • diet;
  • special diet.

Drug therapy can consist of several types of drugs:

  • in the presence of Helicobacter in the stomach, two types of antibiotics are prescribed. They are taken within 10-14 days. If therapy does not help, antibiotics are substituted for others;
  • antacids. Assign to neutralize hydrochloric acid if it is excreted in excess of the gastric mucosa;
  • enzymes are prescribed to normalize the digestive process and restore the microflora of the stomach;
  • enveloping agents are prescribed to regenerate the mucous layer and protect it from external influences;
  • antispasmodics. Relieve spasms of smooth muscles with pain in the abdomen.

Treatment of all types of gastritis, including lymphoid, takes place with the obligatory use of dietary food. Proper nutrition plays a very important role in the healing process, contributing to recovery.

Aggressive food irritates the lining of the stomach and can worsen inflammation. Therefore, fatty, spicy, salty foods are prohibited during gastritis treatment.

It is also necessary to exclude smoked meats, pickles, canned food and flour products from the menu. Carbonated drinks, alcohol and coffee are prohibited.
You need to eat soft and light food that is well digested by the stomach. It includes:

  • boiled vegetables;
  • soups;
  • porridge;
  • mashed potatoes;
  • small varieties of pasta.

You can eat lean meats and fish, boiled, baked or steamed. Fermented milk products, boiled sausage, pates, casseroles are allowed. Recommended drinks green tea, fruit drinks, jelly, compotes.

You also need to adhere to a special diet. You need to eat in small portions, 5 or 6 times during the day. Food must be chewed thoroughly so as not to hinder the work of the stomach. Hot and cold food has a negative effect on the mucous membrane, so food should be at a warm temperature.

Folk remedies

Additionally, you can use various folk remedies to treat gastritis of all types, including lymphoid. Components such as honey vegetable juices, medicinal plants, are inexpensive and affordable remedies that are in every home. They have antibacterial and anti-inflammatory effects and heal the mucous membrane well.

Honey can be eaten in pure form, one to two tablespoons per day, before meals. Honey water also works well. In glass warm water dissolve 15–20 grams of honey. Drink half an hour before meals three times a day.

Propolis tincture is diluted in water, in a ratio of 10 drops per 100 ml. It can be purchased at the pharmacy. You need to drink half a glass before meals for 20-30 minutes. Accept honey water or propolis for two weeks.

Sea buckthorn oil is used as an anti-inflammatory and analgesic agent. It should be drunk one teaspoon half an hour before meals. Blackcurrant juice can be taken with low acidity. Drink half a glass of juice three times a day.

Potato juice has high bactericidal properties. It neutralizes the hydrochloric acid in the stomach. You can drink only freshly squeezed juice from young fruits. Drink the drink immediately after making it. In the same way, you can use squeezed cabbage juice. The compositions are drunk three times a day, in a third of a glass.

Excellent wound healing and bactericidal properties are possessed by infusions and decoctions from medicinal plants... For the preparation of formulations, you can use herbs such as:

  • yarrow;
  • pharmacy chamomile;
  • peppermint;
  • st. John's wort:
  • elecampane;
  • calamus root;
  • celandine.

To prepare a composition with antibacterial and healing effects, take 10 grams of yarrow, chamomile and flax seeds. Pour the ingredients with a glass of boiling water and let it brew for 1–2 hours. Then, the composition should be filtered and taken 2-3 tablespoons before meals.

A good anti-inflammatory pick can be made with calamus root, flax seed, linden blossom, and peppermint leaves. Mix all components and pour 500 ml of hot water. When the mixture is infused, strain it and drink half a glass 30 minutes before meals.

If you are diagnosed with a disease such as lymphoid-type gastritis, do not delay its treatment. Neglected pathologies often take on more severe forms, which are much more difficult to cure.

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Group lymphatic follicles (Peyer's patches) in the form of clusters of lymphoid follicles and diffuse lymphoid tissue are located in the wall of the small intestine, mainly the ileum, in its mucous membrane. The topography of the lymphoid lining and the schematic structure of the lymphatic follicle are shown in Fig. 1.11. and 1.11a.
The formation of Peyer's patches and their anatomical structure in different types animals differ significantly. So, for example, in rodents and in rabbits, Peyer's patches are located at different distances between the ileum and the jejunum, and are formed after 2-4 weeks. postnatal life and function throughout the life of animals. Peyer's patches in ruminants, horses, pigs, dogs and humans are formed in the embryonic period, are located mainly in the wall of the ileum (80-90%), form massive clusters of lymphoid follicles extending anteriorly from the ileocecal angle, which can reach a length 2 meters, for example in ruminants and pigs. Peyer's patches of the ileum of these animals reach their maximum size in young animals and then undergo involution, like thymic tissue.


For example, in sheep, Peyer's plaques of the ileum are formed on the 85th day of embryonic life, their weight at 6 weeks of age is approximately 1% of the body weight, they disappear at 15 months of age and are no longer detected in adult animals. At the same time, the Peyer's patches of the jejunum of these animals function throughout their lives.
Peyer's patches, for example, of sheep, differ in structure. So, Peyer's plaques of the ileum are presented in the form of elongated oval densely packed follicles containing B-lymphocytes and separated from each other by a muff of connective tissue. Peyer's patches of the jejunum consist of pear-like follicles separated by interfollicular tissue and include up to 30% T-lymphocytes.

Structured lymphoid tissue of Peyer's patches
The structured lymphoid tissue of Peyer's patches is conventionally subdivided into three main zones - dome, B-cell and T-cell zones.
The dome is a zone in which both T and B lymphocytes, plasma cells, and macrophages are located. The dome zone is adjacent to the intestinal lumen and is in contact with the epithelium, which covers the plaque and has the ability to transport antigen molecules, including microbial ones, into the lymphoid tissue. This epithelium is called follicle associated epithelium (FAE - Follicule associated epithelium).
The transport function is performed by its specialized M-cells (Fig. 1.12), so named due to the large number of microfolds present on their surface.


The B-cell zone of Peyer's patches is located under the subepithelial dome and includes the lymphatic follicles usually containing the germinal center. Peyer's patches contain 40-45% of B cells, about 20% of B lymphocytes, and expresses IgA on the membrane. The T-cell zone is located around the arteriole, deeper than the B-cell zone. Most of the T cells in this area (about 60%) have a T-helper phenotype (CD4), about 25% of T-lymphocytes have a cytotoxic cell phenotype (CD8). In humans and animals, the overwhelming number of T-lymphocytes of Peyer's patches (they account for 45% of lymphocytes) express the AH-recognizing receptor of the αβ type (\u003e 95%), while γδT-lymphocytes account for only about 5%.
Dendritic cells and monocytes / macrophages of Peyer's patches are less numerous in comparison with other cellular forms and account for 5-10%.
Diffuse lymphoid tissue of Peyer's patches
Diffuse lymphoid tissue of group lymphatic follicles includes two main components - intraepithelial lymphocytes and lamina propria. It is closely associated with follicle-associated epithelium, primarily with its cells - enterocytes. Among enterocytes there is a huge number of intraepithelial lymphocytes - IEL (Intra-Epithelial Lympho-cytes), represented mainly by T-cells (80-90%). For every meter of mucous membrane there are about 1.6x108 such cells. Among the IELs, T-lymphocytes with a phenotype characteristic of both cytotoxic T-lymphocytes and T-helpers are detected. In quantitative composition, IEL T-lymphocytes differ significantly from peripheral blood and from other lymphoid organs. Thus, in the human IEL population, almost all T cells carry the HML-1 marker (Human Mucosal Lymphocyte antigen 1 - antigen 1 of human mucosal lymphocytes) on the membrane. This marker is not expressed on T-lymphocytes of peripheral blood. Moreover, the IEL pool among T-lymphocytes with the CD3 marker contains up to 75% of T-cells of the CDS phenotype (in peripheral blood their number is 20-25%), 6-7% of T-cells of the CD4 phenotype (in peripheral blood, their number is is 35-50%) and up to 40% of γδТ-lymphocytes (in other lymphoid organs their number does not exceed 10%).
Another effector zone of the immune system of the gastrointestinal tract is the lamina propria (L. propria), the main area of \u200b\u200bIgA production in the body. For each Meip of the intestine, there are 10 in 10 cells of this zone that produce immunoglobulins, about 80% of these cells synthesize IgA. The cell composition of L. propria includes T-, B- and NK-lymphocytes, dendritic cells, monocytes, macrophages. In L. propria, eosinophils and mast cells are also found, myofibroblasts (fibroblast-like cells) L, propria form a zone of loose connective tissue that supports its structure.
An important element of the lymphoid tissue of the gastrointestinal mucosa, as well as of the accumulations of lymphoid cells in the mucous membrane of other localizations, are epithelial cells, which, in the course of inflammatory reactions, not only begin to express MHC class II antigens on the membrane and secrete cytokines, but also acquire the ability to represent peptide fragments of AG T-lymphocytes and stimulate their reproduction.
It is believed that lamina propria lymphocytes are similar to peripheral blood lymphocytes in a number of parameters of functional activity. Thus, lymphocytes of such different localizations as peripheral blood and lamina propria express marking structures on the surface in approximately the same proportions, for example, CD4: CD8, equal to 2: 1, and exhibit helper, suppressor, and cytotoxic activity. At the same time, there are significant differences between cells of different localizations. Thus, L. propria B-lymphocytes originate mainly from B1-cells of the abdominal cavity (the bulk of peripheral blood B-lymphocytes are characterized as B2-lymphocytes, the proportion of B-lymphocytes in the blood is about 20%). Moreover, peripheral blood T-lymphocytes do not show helper activity against L. propria B-lymphocytes in the production of secretory IgA. For them, T-helpers are only T-lymphocytes of L. propria. Another important feature of the intestinal mucosa lymphoid tissue is that it (including diffuse lymphoid tissue) contains more T cells than all other lymphoid structures of the body put together.

Solitary (solitary) lymphatic follicles are in the wall small intestine throughout its entire length. Follicles have a small (0.5-3 mm) diameter, lie in the thickness of the intestinal mucosa. In more distal parts of it, they are found in the submucosa and muscle layers. In total, humans have about 15,000 solitary follicles. As a person ages, their number decreases.

Larger intestinal lymphoid formations - peyer's patches - located in the ileum, they are also found in the jejunum and duodenum. In the intestines of children, there are up to 100 of them, in adults - up to 38-40. Peyer's patches are oblong islets protruding into the lumen of the intestine, 2-12 in length and about 1 cm wide. The largest of them are partially located in the submucosa of the intestine.

Peyer's patches - the immunocompetent organs of the human small intestine, actively participating in the recognition of the antigenic structure of the food bolus and the formation of a local, mainly IgA-dependent immune response. Peyer's patches are a collection of individual germinal centers surrounded by dense clusters of lymphocytes. The lymph flowing from Peyer's patches through the lymphatic vessels enters the thoracic lymphatic duct. Above Peyer's patches are the so-called M-cells, which have an uneven surface, cytoplasmic processes surrounding interepal lymphocytes, and have pronounced endocytic activity. Peroxidase introduced into the intestine is found in M \u200b\u200bcells after 5 minutes, and between them and lymphocytes after 1 hour (Ruchti et al, 1980).

Currently, the structure of the intestinal Peyer's plaque has been studied, in which three well-differentiated structural elements have been distinguished: the dome, the follicle itself, and the area dependent on the thymus (Waksman, 1973). The dome is already present in the intestine of the newborn. The degree of its development does not depend on antigenic stimulation. The dome contains B-lymphocytes, which, during the functioning of the follicle, accumulate in the crown adjacent to the dome, can be included in the follicle, where they are stimulated by endotoxins of microorganisms, turning into blast cells. The follicle contains B-lymphocytes, which, interacting with T-lymphocytes during the formation of antibodies, can move to the follicular regions of the mesenteric lymph nodes and to the spleen. The region dependent on the thymus contains T-lymphocytes that are unable to produce antibodies - memory cells, NK cells, T- and B-response helpers, specific killers.


"Immunology of the gastrointestinal tract",
P.M.Sapronenkov


After contact of the body with substances of antigenic nature, a response develops in the form of the formation of antibodies (Iglg A, G, M, D, E), the formation of immune complexes or the reaction of sensitized lymphocytes. There are four main types of allergic reactions (Coombs, Gell, 1975 - cited in: Thompson, 1979). In most cases, in the pathological process occurring in the body, several types of reactions are simultaneously involved ...


Of particular interest are reports on the effect of the thymus on the morphofunctional state of the stomach (Kemileva, 1984). It has been proved that in thymectomized rats morphological changes in the stomach are characterized by hyperemia of the glandular layer of the mucosa, smoothing of its relief, the appearance of mucosal defects - from single point hemorrhages to relatively extensive superficial and deeper erosions. Histological examination of stomach preparations revealed various depths of erosion, dystrophic ...


Among the receptors for the regulation of lymphocytes, the Vi-region of the antibody is especially important. The interaction of antigens and antibodies leads to a modification of the Vi-region of Ig, due to which the latter acquires the ability to bind complement, as well as to attach to certain structures on the cell surface - receptors. T cells can act as both an inducer and an inhibitor of proliferative processes (killer cells) and, thus, enter ...


The organs of the local immune system of the gastrointestinal tract can be considered as effector organs that provide an immune response to contact with antigens entering the body per os from the external environment. It should be emphasized that, despite a certain autonomy of the gastrointestinal tract in the immune response, the nature and severity of the body's response will depend, on the one hand, on the strength of antigenic stimulation, on the other - ...


An important, biologically expedient property of SIgA is its ability, after combining with an antigen, to repel epithelial cells, which occurs due to an increase in the hydrophilicity and electric charge of SIgA (Edebo et al., 1975; Magnusson et al., 1979). Experiments have confirmed that SIgA prevents the fixation of streptococcus to the gum epithelium (Williams, Gibbons, 1972), Vibrio cholerae to the intestinal wall (Freter, 1974)….


Lymphoid gastritis is a chronic type of gastritis of a rare form, found in only one patient out of a hundred diagnosed cases. The disease is an inflammatory and degenerative changes in the gastric mucosa. Under the influence of negative factors, lymphocytes (immune blood cells) leak and accumulate in the tissues, leading to the formation of follicles (growths) on the surface. Therefore, the type of disease is called follicular gastritis.

Follicles that appear in damaged areas can grow to large sizes. They prevent the secretion of gastric juice, disrupt the digestion process and intestinal function.

Clinical studies have established the causes leading to the appearance of the disease.

  • helicobacter pylori (lymphoid, similar to antral gastritis, in the overwhelming majority of cases is caused by bacterial infection);
  • genetic predisposition;
  • improper nutrition;
  • alcohol abuse;
  • smoking;
  • stress.

The listed factors, individually and in combination, create comfortable conditions for the reproduction of Helicobacter. The immune system tries to fight the pathogen by increasing the number of lymphocytes in the affected area. However, chronic processes and the lack of comprehensive treatment lead to the formation of pathological changes.

The manifestation of signs of the disease is weak. There is no severe pain, as opposed to acute forms.

Often, patients come to the doctor with complaints about:

  • slight pain in the upper abdomen, bothering on an empty stomach or after eating;
  • nausea;
  • sour belching;
  • heartburn;
  • unpleasant taste in the mouth;
  • white coating on the tongue;
  • feeling of discomfort and heaviness in the abdomen;
  • violation of the stool.

Symptoms occur infrequently, many seek help in advanced cases. The type of gastritis is dangerous: thickening of the lymphocytic layer and progressive degeneration of mucosal tissues without proper treatment often leads to erosion (with increased acidity) or stomach cancer (with low acidity).

Diagnostics

It is difficult to identify lymphoid gastritis. The difficulty is due to the similarity of symptoms with other types of gastritis.

Several methods are used for diagnosis:

  • Laboratory research. The patient undergoes clinical tests of urine and feces, fecal occult blood test, general blood tests, biochemical blood test, detection of Helicobacter pylori.
  • Endoscopy. The method is known: a flexible tube with a camera at the end is inserted into the stomach through the esophagus. On the monitor, the doctor can see the state of the mucous membrane, the presence and nature of changes.
  • Biopsy. It is done together with endoscopy. A probe is used to take part of the stomach tissue for analysis.
  • Ultrasound procedure. It is carried out in individual cases, it helps to identify the extent of the proliferation of lymphoid tissue.

The use of an endoscopic method allows you to determine that the patient has lymphoid, and not caused by a similar infection with Helicobacter pylori. The variant is localized in and has acute erosive changes in the mucosa.

Treatment

For the treatment of follicular gastritis, like other chronic gastrointestinal diseases, it is necessary to use a set of measures.

Treatment is individual for each case, depending on the clinical picture. Symptoms are not clearly expressed, the situation will gradually worsen, and it will become more difficult to treat the disease.

Drug treatment

If diagnostic studies have confirmed the presence of Helicobacter pylori in the microflora of the gastric mucosa, the doctor first of all prescribes a triple scheme of irradiation (destruction) of the pathogen. Includes an inhibitor (a drug that reduces the production of hydrochloric acid by stomach cells) and two antibiotics.

If the treatment does not work, in addition to the inhibitor, a colloidal drug is prescribed, which creates a film on the damaged areas and stimulates the production of protective mucus. Antibiotics are replaced by others.

Then drugs are prescribed that restore epithelial cells. The doctor may prescribe pain medications.

Unconventional treatments

Treatment folk remedies in combination with medical methods, it gives good results. Before starting therapy, in order to avoid exacerbations, a doctor's consultation is imperative.

In herbal medicine, herbal preparations are recommended to relieve symptoms and chronic manifestations of lymphoid gastritis.

Other methods

Lymphoid gastritis is accompanied by treatment with other products.

  • Plantain juice is useful for the treatment of exacerbations in the gastrointestinal tract. It has an antispasmodic and anti-inflammatory effect, helps to restore the tissues of the mucous membrane. Fresh juice should be drunk at 50 g a quarter of an hour before meals for two weeks.
  • Fresh potato and cabbage juices have a detrimental effect on Helicobacter. The dosage regimen is similar to that of plantain juice.
  • Honey has the ability to lower acidity. It is allowed to use for lymphoid gastritis. It is necessary to dissolve honey (10 g) in a glass of water and drink three times before meals for 20 minutes a day. Alternative medicine advises a decoction: dilute honey in fresh plantain juice (in equal parts). Cook for 20 minutes on low heat, drink before meals for 20 minutes.
  • Possesses antibacterial and anti-inflammatory effects of propolis. For oral administration, a pharmacy tincture is used. For 100 ml of water, 10 drops are taken and drunk before meals for half an hour. The course of admission is two weeks. However, propolis must be used with extreme caution, as the product is considered a strong allergen.
  • Sea buckthorn oil is recommended. Useful material in it they fight bacteria, heal and restore soft tissues. Take 5 ml of oil half an hour before meals.
  • People with decreased secretion are advised to drink juice black currant half a glass three times a day.
  • There are tips to consume fresh leaves and Aloe Arborescent juice. In addition to healing and antimicrobial properties, the plant can stimulate the growth of malignant cells. You will definitely need to consult a doctor when using the plant in treatment.

Diet

In order for lymphoid gastritis to be cured, the number of relapses is minimized, sparing nutrition is shown. When treating acid-dependent gastrointestinal diseases, dishes are conditionally divided into three groups:

  1. Recommended. Lean meat, fish and poultry, soft-boiled eggs or scrambled eggs, soups with lean broths, non-acidic and low-fat dairy products, well-boiled cereals, boiled, baked or steamed vegetables, non-acid fruits are allowed. It is allowed to take not strong coffee and tea, diluted with milk. It is allowed to use marshmallows, marshmallows, milk caramel and ice cream (not on an empty stomach). Bread is best eaten with wheat and slightly dried.
  2. It is recommended to limit. The group includes sausages, salted and canned fish, caviar, spicy and sour soups, sour and fatty dairy products, cheeses. The restriction includes homemade baked goods, corn bread and pasta, spicy and hard-to-digest vegetables, spices, sour fruits and berries, dried fruits, juices and compotes. It is allowed to add butter in small quantities to ready-made cereals and soups, after eating there is a small piece of chocolate or candy.
  3. Not recommended. It is necessary to completely exclude from the diet fatty meats, smoked meats, fried foods, raw eggs, soups and borscht in rich broths, legumes, confectionery (especially with butter creams), spreads, carbonated drinks, alcohol.

A special diet provides split meals from 4 to 6 times a day. Feeling overcrowded after eating, you need to reduce the amount of portions, give preference to mashed dishes, steamed, boiled vegetable and mashed soups. In any case, the diet is prescribed individually and agreed with the attending physician.

Prevention

Chronic diseases differ from acute forms sluggishly current processes formed over the years and habits. To achieve good results in overcoming ailments, including the disease of lymphoid gastritis, preventive measures are necessary.

  • If the cause of the onset of the disease is Helicobacter pylori, all family members should be examined to avoid relapses. If no symptoms are noticeable, the pathogen carrier is likely present.
  • It is necessary to be examined annually by a gastroenterologist.
  • Get rid of bad habits: smoking (especially on an empty stomach), alcohol.
  • Establishing a diet as a habit for life is a guarantee of health digestive system, the whole organism.
  • Follow the diet for a year after establishing a stable remission. It is gradually possible to introduce previously excluded foods into the diet.
  • Moderate physical activity will help restore psycho-emotional balance and start the body's self-healing processes.